Alzheimers & Dementia

Mind Performance Center

J Douglas Brown, DC, DACNB

Neurology & Functional Neurology Practice located in Foley, AL

Today’s medications offer little hope for treating Alzheimer’s disease and dementia because they only target one of many possible problems that contribute to memory loss and cognitive decline. As a specialist in functional neurology, J. Douglas Brown, DC, DACNB, at Mind Performance Center offers treatments that can reverse and potentially prevent dementia. Using customized therapies, Dr. Brown has helped dementia patients return to near-normal functioning. At Mind Performance Center we proudly serve residents of Foley, Fairhope, Daphne, Mobile, and Pensacola. To learn if you’re a good candidate for treatments that target the source of the problem, call the office in Foley, Alabama, or schedule a consultation online.

Alzheimers & Dementia Q & A

What is dementia?

Dementia refers to a group of conditions that cause memory loss and a decline in your cognitive ability that’s severe enough to affect daily functioning. Four primary types of dementia are known to cause progressive brain damage and dysfunction:

  • Alzheimer’s disease
  • Vascular dementia
  • Lewy body dementia
  • Frontotemporal dementia


As the most common type of dementia, Alzheimer’s disease accounts for 60-80% of all cases.

What symptoms develop due to Alzheimer’s and dementia?

Dementia causes symptoms such as:

  • Memory loss
  • Confusion and disorientation
  • Difficulty communicating
  • Finding it hard to concentrate
  • Difficulty with planning and problem solving


Memory loss is usually the first symptom of Alzheimer’s disease and Lewy body dementia. Other symptoms, such as changes in personality, mood swings, and hallucinations, appear as both diseases progress.  

By comparison, the earliest signs of frontotemporal dementia are changes in personality, behavior, and language, with memory loss appearing in later stages.

What treatments improve the symptoms of Alzheimer’s and dementia?

Dr. Brown reviews your medical history and performs a neurological exam, including cognitive testing. He also orders in-depth lab work and evaluates numerous factors that affect the health of your brain, including nutrition, sleep habits, exercise, stress, and medications.

In some patients, Alzheimer’s can be prevented or reversed through a combination of therapies such as:

Bredesen’s ReCODE protocol

Dale E. Bredesen, MD, an internationally recognized expert in neurodegenerative diseases, developed the reversal of cognitive decline protocol, also called the ReCODE protocol.

ReCODE defines all the possible contributors to Alzheimer’s, such as infections, toxins, hormone and nutritional deficiencies, a leaky gut, and metabolic diseases like diabetes. 

There are dozens of potential contributors that all share the same characteristic: They trigger changes in a specific protein, which in turn leads to Alzheimer’s disease.

After identifying your triggers, Dr. Brown develops precision treatments that target every factor affecting your cognitive health. Studies show that following the ReCODE protocol can successfully reverse the cognitive decline of Alzheimer’s and restore your ability to function.

Brain pathway therapies and total brain rehabilitation

Dr. Brown specializes in rehabilitating damaged brain pathways. He trains your brain to form new nerve connections and improves nerve activity by stimulating your central nervous system. Dr. Brown may use sensory, motor, and/or cognitive exercises to stimulate and rehabilitate the dysfunctional pathways in your brain.  

Deep transcranial magnetic stimulation (DTMS)

DTMS rebalances brain activity using magnetic impulses that safely pass through your skull to reach areas of irregular nerve activity in your brain. In addition to potentially improving memory and cognition, DTMS is highly effective for treating depression.

To learn if you’re a good candidate for Dr. Brown’s Alzheimer’s disease and dementia protocols, call Mind Performance Center or schedule an appointment online.